天泉 tiānquán | Source céleste (Nguyen Van Nghi 1971, Lade 1994, Laurent 2000) |
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Tiānshī 天湿 [天濕] (1) | selon Wai Tai Mi Yao (Guillaume 1995), Laurent 2000 |
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Tiānwēn 天温 [天溫] (2) | cité dans le Zhen Jiu Xue ci dian (Guillaume 1995), Laurent 2000 |
Tiānjīng 天泾[天涇] (3) | selon le Tong yi bao tian, ce serait une erreur de transcription (Guillaume 1995) |
Items de localisation
Connexion avec l'organe Coeur
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Objective : To explore the connection pathway between “Tianquan” (PC 2) [2MC] of the Pericardium Meridian and the heart with the combined electrophysiological and morphological techniques. Methods : 34 cats anesthetized with urethane (500 mg/kg) and chloralose (50 mg/kg) were used in the first part of the present study. (I) After isolating the left inferior cardiac nerve (LICN) and the left lateral cutaneous nerve (LLCN) and performing a skin flap bath of paraffin-oil, discharges of LCN were fed to VC-10 oscilloscope and histogramer via a bipolar platinum electrode (reference electrode placed beneath the subcutaneous tissues), and recorded by using a X-Y recorder. LICN was stimulated with an electric stimulator. The myoelectrical activities of “Tianquan” (PC 2) and “Tianfu” (LU 3) areas were recorded respectively through two pairs of acupuncture needle electrodes. The reaction threshold was set to that the electric stimulation just could evoke myoelectric signal changes. (2) In the second part of this study, ten cats anesthetized with 1 % pentobarbital (40 mg/kg) were used. Propidium Iodide (PI, 10 uL) was carefully injected into the sheath of LLCN and the muscles around “Tianquan” (PC 2) and bis Benzimide (bB, 5 uL) injected respectively into and the LICN and the stellate ganglion. Results : Following electric stimulation of LICN, the firing rates of LLCN and myoelectrical activity of“Tianquan” (PC 2) increased pronouncedly in comparison with pre-stimulation (P < 0 - 01); while no significant changes of myoelectric signals of “Tianfu” (LU 3) were found. After cutting off the dorsal and ventral roots of C7 - T2 separately, the thresholds of the discharges of the LLCN and “Tianquan” (PC 2) area responding to stimulation of the LICN increased gradually, being significant in TI, C8 and C7 in comparison with pre-section of nerves. After microinjection of PI and bB, light microscopic examination showed that the single and the double labeled neurons were mainly located in C7, C8 and T, dorsal root ganglions with C8 being the utmost. Conclusion : a) There exists a relatively specific connection between “ Tianquan” (PC 2) and the heart; b) C7 - T1 segments may be responsible for the connection between “Tianquan” (PC 2) of the Pericardium Meridian and the heart.
Acupuncture | Moxibustion | Source |
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Piquer perpendiculairement à 1-2 distances | Roustan 1979 | |
Puncture perpendiculaire entre 0,5 et 1 distance de profondeur | Cautérisation avec 3 à 5 cônes de moxa, moxibustion pendant 5 à 10 minutes | Guillaume 1995 |
Piqûre perpendiculaire de 1 à 1,5 cun | Chauffer 5 à 10 mn | Laurent 2000 |
Sensation de puncture
Sécurité
Classe d'usage | - | point mineur |
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Indication | Association | Source |
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OBJECTIVE: To observe the effect of Electroacupuncture (EA) stimulation of “Tianquan”(PC 2), “Quze” (PC 3), “Neiguan” (PC 6), “Daling” (PC 7) of the Pericardium Meridian on cerebral angiogenesis in cerebral ischemia (CI) rats, so as to reveal its mechanisms underlying improvement of stroke. METHODS: A total of 50 SD rats were equally randomized into normal control, sham, model, EA-Pericardium-Meridian acupoints (EA-PCM) and EA-Lung-Meridian acupoint (EA-LUM) groups. The CI model was established by occlusion of the middle cerebral artery. EA (2-4 V, 20 Hz) was applied to PC 2, PC 3, PC 6, PC 7 and “Tianfu”(LU 3), “Chize” (LU 5), “Lieque” (LU 7), “Taiyuan” (LU 9) of the Lung Meridian for 30 min, once at time-points of 0 h, 6 h, 24 h, 48 h and 72 h, respectively after modeling. Serum nerve growth factor (NGF) and Nogo protein-A (Nogo-A) contents were assayed by enzyme linked immunosorbent assay (ELISA), and cerebral NGF and Nogo-A immunoactivity levels in the ischemic cerebral tissue were detected by immunohistochemistry. RESULTS: (1) Compared to the normal control group, serum NGF and Nogo-A contents, and cerebral NGF immunoactivity level in the model group were significantly increased (P < 0.01). Following EA interventions, serum and cerebral NGF levels were further significantly up-regulated in the EA-PCM and EA-LUM groups (P < 0.01), while serum Nogo-A contents were down-regulated in the two EA groups (P < 0.01). The effect of EA-PCM was markedly superior to that of EA-LUM in up-regulating serum and cerebral NGF levels and down-regulating serum No- go-A level (P < 0.01). No significant differences were found between the normal control and sham groups in serum and cerebral NGF and Nogo-A levels (P > 0.05) , and among the 5 groups in cerebral Nogo-A levels (P > 0.05). CONCLUSION: EA stimulation of acupoints of both Pericardium Meridian and Lung Meridian can up-regulate serum NGF, cerebral NGF expression and down-regulate serum Nogo-A in CI rats, and the effect of Pericardium Meridian is markedly superior to that of Lung Meridian, suggesting a possible better nerve repair effect of EA-PCM acupoints on ischemic brain.